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inflammation
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neuroimmune guidance cues
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Vreeken, D.
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Gils, J.M. van
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Zonneveld, A.J. van
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Neuroimmune guidance cues in vascular (patho)physiology
RIPK1 expression associates with inflammation in early atherosclerosis in humans and can be therapeutically silenced to reduce NF-κB activation and atherogenesis in mice
Downregulation of endothelial plexin A4 under inflammatory conditions impairs vascular integrity
Netrin-4 expression by human endothelial cells inhibits endothelial inflammation and senescence
Comprehensive analysis of neuronal guidance cue expression regulation during monocyte-to-macrophage differentiation reveals post-transcriptional regulation of semaphorin7A by the RNA-binding protein quaking
Prediction power on cardiovascular disease of neuroimmune guidance cues expression by peripheral blood monocytes determined by machine-learning methods
Ephs and ephrins in adult endothelial biology
The identification and function of a Netrin-1 mutation in a pedigree with premature atherosclerosis
EPH receptor B2 stimulates human monocyte adhesion and migration independently of its EphrinB ligands
Endothelial semaphorin 3F maintains endothelial barrier function and inhibits monocyte migration
Netrin-1 and the grade of atherosclerosis are inversely correlated in humans
Disruption of circadian rhythm by alternating light-dark cycles aggravates atherosclerosis development in APOE*3-Leiden.CETP mice
Function and mutation of netrin-1 in premature atherosclerosis
Downregulation of endothelial PLXNA4 under pro-atherosclerotic conditions diminishes vascular integrity enabling monocyte transendothelial migration
Understanding netrins and semaphorins in mature endothelial cell biology
Kidney injury molecule-1 staining in renal allograft biopsies 10 days after transplantation is inversely correlated with functioning proximal tubular epithelial cells
KIM-1 STAINING INTENSITY IN RENAL ALLOGRAFT BIOPSIES 10 DAYS POST TRANSPLANTATION IS INVERSELY CORRELATED WITH FUNCTIONING PROXIMAL TUBULAR EPITHELIAL CELLS
Alternatively spliced tissue factor synergizes with the estrogen receptor pathway in promoting breast cancer progression