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Abatacept in individuals at high risk of rheumatoid arthritis (APIPPRA)
Hyaluronidase treatment of synovial fluid is required for accurate detection of inflammatory cells and soluble mediators
Anticentromere antibody levels and isotypes and the development of systemic sclerosis
Response to: "How to communicate in science" by Klareskog et al
Response to: 'Comment on editorial 'Pathogenic effector functions of ACPA: where do we stand'' by Holmdahl
Neutrophil extracellular traps (NETs) take the central stage in driving autoimmune responses
On the presence of HLA-SE alleles and ACPA-IgG variable domain glycosylation in the phase preceding the development of rheumatoid arthritis
On the presence of HLA-SE alleles and ACPA-IgG variable domain glycosylation in the phase preceding the development of rheumatoid arthritis
Pathogenic effector functions of ACPA: Where do we stand?
Anti-cyclic citrullinated peptide antibodies, other common autoantibodies, and smoking as risk factors for lymphoma in patients with rheumatoid arthritis
Masscytometry identifies CD8 T-cell diversity in human atherosclerotic lesions
The extensive glycosylation of the ACPA variable domain observed for ACPA-IgG is absent from ACPA-IgM
The extensive glycosylation of the ACPA variable domain observed for ACPA-IgG is absent from ACPA-IgM
Antibodies against collagen type II are not a general marker of acute arthritis onset
ADRENIC ACID AS A NOVEL ANTI-INFLAMMATORY PLAYER IN OSTEOARTHRITIS
THE ROLE OF POST-TRANSLATIONAL MODIFICATION AND AUTOREACTIVITY
ASSOCIATION BETWEEN CONVERSION TO ACPA/RF SERONEGATIVE STATUS AND CLINICAL OUTCOMES FOLLOWING TREATMENT WITH ABATACEPT IN COMBINATION WITH METHOTREXATE COMPARED WITH METHOTREXATE ALONE IN PATIENTS WITH EARLY RHEUMATOID ARTHRITIS AND POOR PROGNOSTIC INDICA
THE IMPACT OF THERAPY ON ANTI-CARBAMYLATED PROTEIN ANTIBODY ISOTYPES AND SEROSTATUS IN PATIENTS WITH EARLY RA TREATED WITH ABATACEPT AND METHOTREXATE
IN THE ELDERLY ACPA-NEGATIVE RA IS MORE PREVALENT THAN ACPA-POSITIVE RA WHILE THE COMPOSITION OF THE ACPA-RESPONSE APPEARS IDENTICAL
ANTISENSE LONG NONCODING RNAS ARE DEREGULATED IN SKIN TISSUE OF SSC PATIENTS
MAST CELLS SHOW A REPROGRAMMED TRANSCRIPTIONAL SIGNATURE FOLLOWING REPEATED IGG STIMULATIONS
BREACH OF AUTOREACTIVE B-CELL TOLERANCE BY
LACK OF OBESITY-RELATED FEATURES IN ADIPOCYTES AND INFLAMMATORY CELLS IN THE INFRAPATELLAR FAT PAD (IFP)
PATHOPHYSIOLOGIC PROCESSES LEADING TO THE DEVELOPMENT OF AUTOIMMUNITY
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