Leiden University Scholarly Publications

Properdin binds independent of complement activation in an in vivo model of anti-glomerular basement membrane disease

Properdin and factor H production by human dendritic cells modulates their T-cell stimulatory capacity and is regulated by IFN-

Monomeric C-reactive protein inhibits renal cell-directed complement activation mediated by properdin

Functional assessment of mouse complement pathway activities. and quantification of C3b/C3c/iC3b in an experimental model of mouse renal ischaemia/reperfusion injury

Myeloperoxidase Directs Properdin-Mediated Complement Activation

Differential Complement Activation Pathways Promote C3b Deposition on Native and Acetylated LDL thereby Inducing Lipoprotein Binding to the Complement Receptor 1

Phagocytosis of apoptotic or necrotic cells differentially regulates the transcriptional expression of IL-12 family members in dendritic cells

Properdin-dependent activation and control of immune-homeostasis and autoimmunity

Neutrophil components as regulators of the complement system

Dendritic cells express alternative pathway components properdin and factor H: Differential regulation by Interferons and IL-27

Nucleosomes and C1q bound to glomerular endothelial cells serve as targets for autoantibodies and determine complement activation