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sunitinib
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Monitoring intracellular tacrolimus concentrations and its relationship with rejection in the early phase after renal transplantation
Pre-transplant donor-reactive IL-21 producing T cells as a tool to identify an increased risk for acute rejection
Avoiding tacrolimus underexposure and overexposure with a dosing algorithm for renal transplant recipients
Banff lesions and renal allograft survival in chronic-active antibody mediated rejection
Characterization of donor and recipient CD8+tissue-resident memory T cells in transplant nephrectomies
Chronic-active antibody-mediated rejection with or without donor-specific antibodies has similar histomorphology and clinical outcome - a retrospective study
Donor-Derived Cell-Free DNA as Minimally Invasive Tool to Diagnose Acute Rejection after Kidney Transplantation
Elevated intragraft expression of innate immunity and cell death-related markers is a risk factor for adverse graft outcome
Elevated intragraft expression of innate immunity and cell death-related markers is a risk factor for adverse graft outcome
Pretransplant Numbers of CD16(+) Monocytes as a Novel Biomarker to Predict Acute Rejection After Kidney Transplantation: A Pilot Study
A Randomized Controlled Clinical Trial Comparing Belatacept With Tacrolimus After De Novo Kidney Transplantation
Donor Genotype and Intragraft Expression of CYP3A5 Reflect the Response to Steroid Treatment During Acute Renal Allograft Rejection
Primary Polyomavirus Infection, Not Reactivation, as the Cause of Trichodysplasia Spinulosa in Immunocompromised Patients
Effect of high salt diet on blood pressure and renal damage during vascular endothelial growth factor inhibition with sunitinib
AN ACUTE CELLULAR REJECTION WITH DETRIMENTAL OUTCOME OCCURRING UNDER BELATACEPT-BASED IMMUNOSUPPRESSIVE THERAPY: AN IMMUNOLOGICAL ANALYSIS
Greater Sensitivity of Blood Pressure Than Renal Toxicity to Tyrosine Kinase Receptor Inhibition With Sunitinib
Follicular T helper cells and humoral reactivity in kidney transplant patients
DONOR GENOTYPE AND INTRAGRAFT EXPRESSION OF CYP3A5 REFLECT THE RESPONSE TO STEROID TREATMENT DURING ACUTE RENAL ALLOGRAFT REJECTION